“…The mammalian gastrointestinal mucosa is a rapidly self-renewing tissue in the body, and its homeostasis is preserved through the strict regulation of epithelial cell proliferation, growth arrest, and apoptosis. …”

Tabla de Contenidos: “…Consciousness, Emotional Self-Regulation and the Brain -- Editorial page -- Title page -- LCC page -- Table of contents -- List of contributors -- Introduction -- Overview of chapter contents -- Acknowledgements -- Emotion self-regulation -- Introduction -- Components of emotion -- Gross' process model of emotion self-regulation -- Theoretical and empirical challenges in measuring antecedent emotion self-regulation -- Izard's multi-system model of emotion generation -- Implications of Izard's multi-system model of emotion activation for emotion self-regulation -- Emotion self-regulation and emotional homeostasis -- A sequential model of emotion self-regulation -- Control regulation -- Emotional dissociation -- Enhancing and suppressing emotional expression -- Anticipatory regulation -- Exploratory regulation -- Emotion self-regulation as a contextually-bound process -- Summary and future research directions -- References -- Temperament and emotional regulation -- Introduction -- Theoretical and empirical approaches to emotional regulation -- Emotional regulation in early childhood: Normative development -- Temperament and early emotional development -- Temperament and the development of emotional regulation -- Direct effects of temperament -- Moderated effects of temperament -- Mediational effects of temperament -- Future directions in the study of temperament and emotional regulation -- Summary -- Acknowledgements -- References -- Emotion dysregulation and psychopathology -- Introduction -- Functionalist theory -- Emotion regulation and dysregulation -- What gets regulated in emotion regulation? …”

Tabla de Contenidos: “…-Los mecanismos de regulación y la homeostasis de los geosistemas.; 9.2- La resiliencia y la estabilidad de los paisajes:; 9.3.…”

Tabla de Contenidos: “…INTRODUCCIÓN; 2.2. CONCEPTO DE HOMEOSTASIS…”

Tabla de Contenidos: “…DISTRIBUCIÓN DEL AGUA EN EL CUERPO -- 1.3. LA DESEADA HOMEOSTASIS HÍDRICO-ELECTROLÍTICA -- 1.4. INGRESOS DE AGUA -- 1.5. …”

Tabla de Contenidos: “…Intro -- Cover -- Title Page -- Copyright Page -- Preface -- Acknowledgments -- Contents -- Chapter 1 THE HUMAN BODY: AN ORIENTATION -- An Overview of Anatomy and Physiology -- Levels of Structural Organization -- Maintaining Life -- Homeostasis -- The Language of Anatomy -- At the Clinic -- The Finale: Multiple Choice -- Chapter 2 BASIC CHEMISTRY -- Concepts of Matter and Energy -- Composition of Matter -- Molecules, Chemical Bonds, and Chemical Reactions -- Biochemistry: The Composition of Living Matter -- Incredible Journey: A Visualization Exercise for Biochemistry -- At the Clinic -- The Finale: Multiple Choice -- Chapter 3 CELLS AND TISSUES -- Cells -- Overview -- Anatomy of a Generalized Cell -- Cell Physiology -- Body Tissues -- Tissue Repair -- Developmental Aspects of Cells and Tissues -- Incredible Journey: A Visualization Exercise for the Cell -- At the Clinic -- The Finale: Multiple Choice -- Chapter 4 SKIN AND BODY MEMBRANES -- Classification of Body Membranes -- Integumentary System (Skin) -- Basic Functions of the Skin -- Basic Structure of the Skin -- Appendages of the Skin -- Homeostatic Imbalances of the Skin -- Developmental Aspects of the Skin and Body Membranes -- Incredible Journey: A Visualization Exercise for the Skin -- At the Clinic -- The Finale: Multiple Choice -- Chapter 5 THE SKELETAL SYSTEM -- Bones-An Overview -- Axial Skeleton -- Skull -- Vertebral Column -- Thoracic Cage -- Appendicular Skeleton -- Bone Fractures -- Joints -- Homeostatic Imbalances of Bones and Joints -- Developmental Aspects of the Skeleton -- Incredible Journey: A Visualization Exercise for the Skeletal System -- At the Clinic -- The Finale: Multiple Choice -- Chapter 6 THE MUSCULAR SYSTEM -- Overview of Muscle Tissues -- Microscopic Anatomy of Skeletal Muscle -- Skeletal Muscle Activity -- Muscle Movements, Types, and Names…”

Tabla de Contenidos:

Tabla de Contenidos: “…PREVIOUS RECOMMENDATIONS FROM PROFESSIONAL ORGANIZATIONS -- USAGE PATTERNS -- SCOPE OF REPORT -- UNDERLYING ASSUMPTIONS -- ORGANIZATION OF THE REPORT -- References -- 12 Assessment of Nutrient Needs -- NUTRIENT HOMEOSTASIS -- ASSESSMENT OF NUTRITIONAL STATUS -- ASSESSMENT OF NUTRIENT NEEDS -- ASSESSMENT OF DIETARY ADEQUACY -- RECOMMENDED DIETARY ALLOWANCES -- FACTORS INFLUENCING NUTRIENT REQUIREMENTS AND THE NEED FOR SUPPLEMENTATION -- NUTRIENT-NUTRIENT INTERACTIONS -- NUTRIENT TOXICITIES -- SUMMARY -- CLINICAL IMPLICATIONS -- References -- 13 Dietary Intake During Pregnancy -- AVAILABLE DIETARY DATA -- LIMITATIONS OF THE DATA -- NUTRIENT INTAKES OF PREGNANT WOMEN -- ASSOCIATIONS OF MATERNAL CHARACTERISTICS WITH NUTRIENT INTAKE DURING PREGNANCY -- Ethnic Background -- Socioeconomic and Demographic Factors -- WIC Participation -- Maternal Age -- SUMMARY -- References -- 14 Iron Nutrition During Pregnancy -- IMPORTANCE OF IRON DURING PREGNANCY -- Definition of Anemia and Iron Deficiency -- Effects of Maternal Anemia on the Newborn -- PREVALENCE OF IRON DEFICIENCY -- SPECIAL GROUPS AT RISK -- Socioeconomic Indicators -- Racial and Ethnic Backgrounds -- Adolescents -- IRON METABOLISM IN RELATION TO PREGNANCY -- Essential and Storage Iron -- Iron Loss -- Regulation of Body Iron -- Absorption of Nonheme and Heme Iron from Food -- Intestinal Regulation -- IRON REQUIREMENTS FOR PREGNANCY -- CHARACTERISTICS OF IRON DEFICIENCY DURING PREGNANCY -- Laboratory Characteristics of Impaired Hemoglobin Production -- Physiologic Changes in Hemoglobin Concentration and Criteria for Anemia During Pregnancy -- ABSORPTION OF IRON SUPPLEMENTS -- Absorption from a Test Dose -- Therapeutic Trials -- How Much Extra Iron Must Be Absorbed from a Supplement in Order To Prevent Iron Deficiency? …”

Tabla de Contenidos: “…DiPiro -- Chapter 73: Acid-Base Disorders -- Chapter 74: Acute Kidney Injury -- Chapter 75: Chronic Kidney Disease -- Chapter 76: Electrolyte Homeostasis -- SECTION 15: RESPIRATORY DISORDERS Edited by Terry L. …”

Tabla de Contenidos: “…4.1 The RtcA RNA Cyclase -- 4.2 The RtcB RNA Ligase -- 4.3 The RtcB RNA Ligase in Prokaryotes -- 4.4 The RtcB RNA Ligase in Metazoans -- 5 The Bacterial Rtc RNA Repair System -- 5.1 Diversity of the rtc Operon in Bacteria -- 5.2 The RtcR Transcriptional Regulator and Its CARF Domain -- 5.3 Expression and Function of the Rtc System in Bacteria -- 6 Conclusions -- References -- Part II: RNA Damage and Repair in Eukaryotes -- Oxidative and Nitrative RNA Modifications in Plants -- 1 Introduction -- 2 Oxidative Modifications of RNA in Plants -- 3 Nitrative Modifications of RNA in Plants -- 4 Conclusions -- References -- The Role of Ribonucleases in RNA Damage, Inactivation and Degradation -- 1 Introduction -- 2 Quality Control Mechanisms to Manage Damaged RNA -- 3 Ribosome Quality Control (RQC) and No-Go-Decay (NGD) -- 4 Xrn1, a 5'-3' Exoribonuclease, and the Exosome, a 3'-5' Exoribonuclease Complex -- 5 Cue2 as a Potential NGD Endonuclease -- 6 The Role of Ribonucleases in Targeting and Inactivating Host and Foreign RNA -- 7 Regulatory RNase 1 (Regnase-1) -- 8 NEDD4-Binding Protein 1 (N4BP1) -- 9 Zinc Finger Antiviral Protein (ZAP) and Co-Factor Nucleases -- 10 ZAP and KHNYN -- 11 ZAP and the Exosome Complex -- 12 The 2',5'-Oligoadenylate Synthetase (OAS)/RNAse L System -- 13 Conclusion -- References -- Cytoplasmic mRNA Recapping: An Unexpected Form of RNA Repair -- 1 The Nature of the Cap and Its Role in mRNA Metabolism -- 2 Decapping and 5' Decay -- 3 Early Evidence for Uncapped and Recapped Transcripts -- 4 The Discovery of Cytoplasmic Capping -- 5 The Proteins of the Cytoplasmic Capping Complex -- 6 Cap Homeostasis as an RNA Repair Mechanism That Modulates Translation, mRNA Decay, and Translational Control -- 7 Is There a Relationship Between Recapped Ends and CAGE Tags? …”

Tabla de Contenidos: “…Overview of Violations and Noncompliance -- Key Points -- 7.1 Human error -- Models of Human Error -- The Skills, Rules, Knowledge Framework -- Slips, Lapses, and Mistakes -- Human Information Processing Model -- Model of Situational Awareness -- Human Errors in the Context of a Flawed System -- Prevalence of Human Error -- Performance Shaping Factors -- Human Limits and Susceptibility to Bias -- Potential Solutions for Managing Human Error -- Summary: Human Error -- Key Points -- References -- 7.2 Intentional noncompliance -- Recognizing Intentional Noncompliance -- Tools and Methods for Analyzing Noncompliant Behaviors -- Work Compatibility Model -- ABC Model -- Theory of Planned Behavior -- The Links Between Noncompliance and Safety Culture -- Risk Homeostasis and Safety Noncompliance -- Actions to Reduce Intentional Noncompliances -- Summary: Intentional Noncompliance -- Key Points -- References -- References -- 8 Human factors in incident investigation -- List of Abbreviations -- The Investigation Process -- Key Stages of an Investigation -- Investigation Methods -- Advantages of Mainstream Methods -- Disadvantages of Mainstream Methods -- Choice of Method -- Including Human Factors Within Existing Investigation Methods -- Human Factors Considerations for Investigators -- Key Qualities of Investigators -- Interviewing Skills -- Cognitive Bias in the Investigation Process -- Managing Investigation Capability -- Proactive Application of Human Factors Analysis -- Summary: Human Factors in Incident Investigation -- Key Points -- References -- III. …”

Tabla de Contenidos: “…Summary -- Introduction -- 1.1 Protein aggregation and amyloidoses -- 1.1.1 Mechanisms of amyloid fibril formation -- 1.1.2 Oligomer structural polymorphism: fibrillar and prefibrillar oligomers -- 1.1.3 Oligomer toxicity: common mechanism of pathogenesis -- 1.1.4 Amyloid formation is an inherent property of polypeptide chains: functional amyloid and disease unreleated amyloid -- 1.2 HypF-N: model protein of amyloid aggregation unrelated to disease -- 1.2.1 Function, structure and aggregation of HypF-N -- 1.2.2 HypF-N protofibrils interact with cell membranes originating a cytotoxic cascade -- 1.2.3 A causative link between the structure of HypF-N oligomers and their ability to cause cellular dysfunction -- 1.3 Alzheimer's disease -- 1.3.1 The Alzheimer phenotype -- 1.3.2 The elaborate processing of APP -- 1.3.3 The genetics of Alzheimer's disease -- 1.3.4 Peripheral cells as a tool to identify and test hypotheses on AD pathophysiology -- 1.3.5 Adult neurogenesis and stem cell technology for AD -- 1.4 Cholesterol and gangliosides in the central nervous system (CNS) -- 1.4.1 Brain cholesterol metabolism -- 1.4.2 Ganglioside metabolism -- 1.4.3 Lipid rafts -- 1.4.4 Role of cholesterol in AD -- 1.4.5 Role of gangliosides in AD -- 1.5 Aim of the study -- Materials & Methods -- 2.1 Materials -- 2.1.1 Chemicals -- 2.1.2 Fluorescent probes -- 2.1.3 Peptides and aggregation protocols -- 2.2 Cell cultures -- 2.3 Methods -- 2.3.1 Separation processes -- 2.3.2 Differentiation of human mesenchymal stromal cells -- 2.3.3 Modulation of membrane cholesterol levels -- 2.3.4 Modulation of membrane GM1 levels -- 2.3.5 Cholesterol content measurements -- 2.3.6 GM1 content measurements -- 2.3.7 Cell exposure to peptide aggregates -- 2.3.8 Analysis of aggregate interaction with the cells -- 2.3.9 Analysis of aggregate interaction with GM1 -- 2.3.10 Analysis of aggregate internalisation -- 2.3.11 Analysis of membrane permeability -- 2.3.12 Analysis of cytosolic Ca2+ dyshomeostasis -- 2.3.13 Evaluation of ROS production -- 2.3.14 Analysis of lipid peroxidation -- 2.3.15 Cytotoxicity assay and cell death analysis: apoptotic and necrotic markers -- 2.3.16 Steady-state fluorescence anisotropy -- 2.3.17 Atomic force microscopy (AFM) -- 2.3.18 Measurements of the fluorescence intensities -- 2.3.19 Statistical analysis -- Results -- 3.1 Results I -- 3.1.1 A protective role for lipid raft cholesterol against amyloidinduced membrane damage in human neuroblastoma cells -- 3.1.2 Aß42 oligomer binding to the cell surface and its cytotoxic effect are modulated by membrane cholesterol content -- 3.1.3 Aß42 oligomers colocalize with lipid rafts -- 3.1.4 Isolation and characterization of DRMs -- 3.1.5 Effects of ADDLs on lipid raft structural order -- 3.1.6 AFM imaging of supported DRMs purified from cells exposed to ADDLs -- 3.2 Results II -- 3.2.1 Lipid rafts mediate amyloid-induced calcium dyshomeostasis and oxidative stress in Alzheimer's disease -- 3.2.2 Lipid rafts are primary interaction sites for Aß42 oligomers at the plasma membrane -- 3.2.3 Cholesterol and GM1 mediate Aß42 accumulation at the plasma membrane -- 3.2.4 Cholesterol and GM1 mediate Ca2+ dyshomeostasis and extensive membrane permeabilization induced by Aß42 oligomers -- 3.2.5 GM1 modulates lipid peroxidation and cytotoxicity induced by Aß42 oligomers -- 3.2.6 GM1 mediates Aß42-induced Ca2+ dyshomeostasis, lipid peroxidation and cytotoxicity in rat cortical neurons -- 3.3 Results III -- 3.3.1 Membrane lipid composition and its physicochemical properties define cell vulnerability to aberrant protein oligomers -- 3.3.2 Membrane cholesterol content modulates oligomer cytotoxicity -- 3.3.3 Membrane cholesterol modulates oligomer-induced alteration of intracellular Ca2+ homeostasis and ROS levels -- 3.3.4 Cholesterol levels modulate membrane permeability to the oligomers -- 3.3.5 Membrane GM1 affects the cytotoxic and permeabilizing effects of HypF-N oligomers -- 3.3.6 GM1, rather than cholesterol, plays a dominant role in oligomer cytotoxicity and membrane permeability -- 3.4 Results IV -- 3.4.1 Neuronal differentiation of human mesenchymal stromal cells increases their resistance to Aß42 aggregate toxicity -- 3.4.2 Neuronal differentiation of hMSCs results in reduced levels of membrane GM1 -- 3.4.3 Neuronal differentiation of hMSCs reduces the interaction of Aß42 oligomers with the cell surface -- 3.4.4 Neuronal differentiation of hMSCs reduces Aß42 oligomerinduced intracellular Ca2+ dyshomeostasis and oxidative stress -- 3.4.5 Neuronal differentiation of hMSCs increases cell resistance to Aß42 aggregates Discussion -- 4.1 A protective role for lipid raft cholesterol against amyloid-induced membrane damage in human neuroblastoma cells -- 4.2 Lipid rafts mediate amyloid-induced calcium dyshomeostasis and membrane permeabilization in Alzheimer's fibroblasts -- 4.3 Membrane lipid composition and its physicochemical properties define cell vulnerability to aberrant protein oligomers -- 4.4 Neuronal differentiation of human mesenchymal stromal cells increases their resistance to Aß42 aggregate toxicity -- 4.5 Concluding remarks -- Abbreviations -- References…”

Tabla de Contenidos: “…The Relationships Between Free Energy, the Equilibrium State, and Nonequilibrium Concentrations of Reactants and Products -- Equilibrium, Le Chatelier's Principle, and the Standard State -- Changes in Concentration and .G -- .G versus .G°, Q versus K, and Homeostasis versus Equilibrium -- Water, H+ in Buffered Solutions, and the "Biochemical Standard State" -- 3.4. …”

Tabla de Contenidos: “…What are the Requirements for Life? -- 1.4. Homeostasis is Maintained by Negative Feedback -- 1.5. …”

Tabla de Contenidos: “…HIF, principal regulador de la homeostasis del oxígeno --3. Estructura del HIF1 --4. …”
Enlace del recurso

Tabla de Contenidos: “…Ormsbee, Vincent Miller, Caitlin Sheridan and Feng He / Protein-Pacing and Multi-Component Exercise Training Improves Physical Performance Outcomes in Exercise-Trained Women: The PRISE 3 Study, Reprinted from: Nutrients 2016, 8(6), 332; doi: 10.3390/nu8060332 -- Mauricio Castro-Sepulveda, Neil Johannsen, Sebastián Astudillo, Carlos Jorquera, Cristian Álvarez, Hermann Zbinden-Foncea and Rodrigo Ramírez-Campillo Effects of Beer, Non-Alcoholic Beer and Water Consumption before Exercise on Fluid and / Electrolyte Homeostasis in Athletes, Reprinted from: Nutrients 2016, 8(6), 345; doi: 10.3390/nu8060345 -- Kelly Pritchett, Robert Pritchett, Dana Ogan, Phil Bishop, Elizabeth Broad and Melissa LaCroix / 25(OH)D Status of Elite Athletes with Spinal Cord Injury Relative to Lifestyle Factors, Reprinted from: Nutrients 2016, 8(6), 374; doi: 10.3390/nu8060374 -- Malgorzata Michalczyk, Milosz Czuba, Grzegorz Zydek, Adam Zajac and Józef Langfort / Dietary Recommendations for Cyclists during Altitude Training, Reprinted from: Nutrients 2016, 8(6), 377; doi: 10.3390/nu8060377 -- Daniel König, Denise Zdzieblik, Anja Holz, Stephan Theis and Albert Gollhofer / Substrate Utilization and Cycling Performance Following PalatinoseTM Ingestion: A Randomized, Double-Blind, Controlled Trial, Reprinted from: Nutrients 2016, 8(7), 390; doi: 10.3390/nu8070390 -- Daniel A. …”

“…The purpose of this research topic is to review the role of the LECs during immune homeostasis and cancer. Considering the critical role of lymphangiogenesis in many pathologies like chronic and acute inflammation, autoimmunity, wound healing, graft rejection, and tumor metastasis, it is important to understand the molecular mechanisms that govern the cross talks between the LECs and immune cells during homeostasis and inflammation…”

“…The impact of the cellular microenvironment, including local production of ACTH is discussed, both as an important factor in the maintenance of homeostasis, but also in pathological situations, such as severe inflammation. …”

“…This homeostasis, coupled with highly conserved capping proteins, is sufficient for protection. …”

“…The impact of BIM on B cell homeostasis is discussed by Liu et al. The role of the MEK-ERK pathway in B cell tolerance is discussed by Greaves et al. and the transcriptional regulation of germinal center development is reviewed by Song and Matthias. …”