Tabla de Contenidos: “…2.4 Probability Theory Predicts Mendelian Ratios -- The Product Rule -- Experimental Insight 2.2 -- The Sum Rule -- Conditional Probability -- Experimental Insight 2.3 -- Binomial Probability -- 2.5 Chi-Square Analysis Tests the Fit between Observed Values and Expected Outcomes -- The Normal Distribution -- Chi-Square Analysis -- Chi-Square Analysis of Mendel's Data -- 2.6 Autosomal Inheritance and Molecular Genetics Parallel the Predictions of Mendel's Hereditary Principles -- Autosomal Dominant Inheritance -- Autosomal Recessive Inheritance -- Molecular Genetics of Mendel's Traits -- Case Study: Inheritance of Sickle Cell Disease in Humans -- Summary -- Keywords -- Problems -- Chapter 3: Cell Division and Chromosome Heredity -- 3.1 Mitosis Divides Somatic Cells -- Stages of the Cell Cycle -- Substages of M Phase -- Chromosome Distribution -- Completion of Cell Division -- Cell Cycle Checkpoints -- Cell Cycle Mutations and Cancer -- 3.2 Meiosis Produces Gametes for Sexual Reproduction -- Meiosis versus Mitosis -- Meiosis I -- Meiosis II -- The Mechanistic Basis of Mendelian Ratios -- Segregation in Single-Celled Diploids -- 3.3 The Chromosome Theory of Heredity Proposes that Genes are Carried on Chromosomes -- Genetic Analysis 3.1 -- X-Linked Inheritance -- Testing the Chromosome Theory of Heredity -- 3.4 Sex Determination Is Chromosomal and Genetic -- Sex Determination in Drosophila -- Genetic Analysis 3.2 -- Mammalian Sex Determination -- Diversity of Sex Determination -- Experimental Insight 3.1 -- 3.5 Human Sex-Linked Transmission Follows Distinct Patterns -- Expression of X-Linked Recessive Traits -- Genetic Analysis 3.3 -- X-Linked Dominant Trait Transmission -- Y-Linked Inheritance -- 3.6 Dosage Compensation Equalizes the Expression of Sex-Linked Genes -- Case Study: The (Degenerative) Evolution of the Mammalian Y Chromosome -- Summary…”

“…The book is a unique collection of research on sign languages that have emerged in rural communities with a high incidence of, often hereditary, deafness. These sign languages represent the latest addition to the comparative investigation of languages in the gestural modality, and the book is the first compilation of a substantial number of different "village sign languages". …”

“…As a result of the death of Duke Charles the Bold of Burgundy and the marriage of his only daughter Mary to Maximilian, the heir of Emperor Frederick III, the economically and culturally prospering lands of Flanders and Brabant became hereditary possessions of the Habsburgs. With this, the foundations for the Habsburgs's hegemony were laid. …”

Tabla de Contenidos: “…Plantejament general sobre les substitucions hereditàries al Llibre IV del CCCat (capítol V del Títol II); 2. …”

Tabla de Contenidos: “…Victor Hoffbrand and Ralph Green -- Haemoglobin and the inherited disorders of globin synthesis / David Weatherall -- Sickle cell anaemia / Elliott Vichinsky -- Hereditary disorders of the red cell membrane / Edward Gordon-Smith -- Red cell metabolic disorders / Edward Gordon-Smith -- Acquired haemolytic anaemias / Edward Gordon-Smith and Judith C.W. …”

Tabla de Contenidos: “…Fanconi anemia and other hereditary Bone Marrow Failure syndromes -- 79. Hemoglobinopathies (Sickle Cell Disease and Thalassemia) -- 80. …”

“…Together, these approaches capture the so-called true concurrency based on truly concurrent bisimilarities, such as pomset bisimilarity, step bisimilarity, history-preserving (hp-) bisimilarity and hereditary history-preserving (hhp-) bisimilarity. This book provides readers with all aspects of algebraic theory for localities, including the basis of semantics, calculi for static localities, axiomatization for static localities, as well as calculi for dynamic localities and axiomatization for dynamic localities. …”

Tabla de Contenidos: “…Immunodeficient Rodents -- Copyright -- Preface -- Contents -- 1 Introduction -- IMMUNE SYSTEM FUNCTION -- Host Defense Systems -- The Nature of Adaptive Immunity -- Immunologic Specificity -- Afferent, Central, and Efferent Limbs of the Immune System -- Afferent Limb -- Central Limb -- Efferent Limb -- Immunodeficiency -- EFFECT OF ENVIRONMENTAL FACTORS ON IMMUNE FUNCTION -- Noninfectious Agents -- Infectious Agents -- GENERAL CONSIDERATIONS FOR MAINTAINING IMMUNODEFICIENT RODENTS -- MUTATIONS -- GENE MARKERS AND CHROMOSOME MAPS -- NOMENCLATURE AND SOURCES OF IMMUNODEFICIENT RODENTS -- General Reading -- 2 Hereditary Immunodeficiencies -- MICE WITH SINGLE MUTATIONS -- Bcgr, Bcgs (Resistance and Susceptibility to Mycobacterium bovis) -- Ityr, Itys (Resistance and Susceptibility to Salmonella... -- Genetics -- Pathophysiology -- Husbandry -- Reproduction -- bg (Beige) -- Genetics -- Pathophysiology -- Husbandry -- Reproduction -- db (Diabetes) -- Genetics -- Pathophysiology -- Husbandry -- Reproduction -- df (Ames Dwarf) -- Genetics -- Pathophysiology -- Husbandry -- Reproduction -- Dh (Dominant Hemimelia) -- Genetics -- Pathophysiology -- Husbandry -- Reproduction -- dw (Dwarf) -- Genetics -- Pathophysiology -- Husbandry -- Reproduction -- gl (Grey-Lethal) -- Genetics -- Pathophysiology -- Husbandry -- Reproduction -- gld (Generalized Lymphoproliferative Disease) -- Genetics -- Pathophysiology -- Husbandry -- Reproduction -- Hc0 (Hemolytic Complement Absent) -- Genetics -- Pathophysiology -- Husbandry -- Reproduction -- hr (Hairless) -- hrrh (Rhino) -- Genetics -- Pathophysiology -- Husbandry -- Reproduction -- lh (Lethargic) -- Genetics -- Pathophysiology -- Husbandry -- Reproduction -- lpr (Lymphoproliferation) -- Genetics -- Pathophysiology -- Husbandry -- Reproduction -- Lpsd (Lipopolysaccharide Response, Defective) -- Genetics…”

“…En nuestra población, contrariamente a lo publicado, la mutación sí se segregó con la enfermedad, y todos los miembros con diagnóstico clínico y de imagen de PQRAD presentaron dicha mutación Autosomal dominant polycystic kidney disease is a multiorganic hereditary disorder. It is responsible for 7-10 % of cases of end stage renal failure. …”
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Tabla de Contenidos: “…PART I: Basic Science of Pulmonary Development and Pulmonary Arterial Disease -- 1 Perspective for Part I -- 2 The alveolar stem cell niche of the mammalian lung -- 3 Lung development and Notch signalling -- 4 Specialized smooth muscle cell progenitors in pulmonary hypertension -- 5 Diverse Pharmacology of Prostacyclin Mimetics: Implications for Pulmonary Hypertension -- 6 Endothelial-to-mesenchymal transition in pulmonary hypertension -- 7 Extracellular vesicles, MicroRNAs and Pulmonary Hypertension -- 8 Roles of Tbx4 in the lung mesenchyme for airway and vascular development -- 9 A lacZ reporter transgenic mouse line revealing the development of pulmonary artery -- 10 Roles of stem cell antigen-1 in the pulmonary endothelium -- 11 Morphological characterization of pulmonary microvascular disease in bronchopulmonary dysplasia caused by hyperoxia in newborn mice -- 12 Involvement of CXCR4 and stem cells in a rat model of pulmonary arterial hypertension -- 13 Ca2+ signal through inositol trisphosphate receptors for cardiovascular development and pathophysiology of pulmonary arterial hypertension -- PART II: Abnormal pulmonary circulation in the developing lung and heart -- 14 Perspective for Part II -- 15 Pathophysiology of Pulmonary Circulation in Congenital Heart Disease -- 16 Development of Novel Therapies for Pulmonary Hypertension by Clinical Application of Basic Research -- 17 Using Patient-Specific Induced Pluripotent Stem Cells to Understand and Treat Pulmonary Arterial Hypertension -- 18 Modeling pulmonary arterial hypertension using induced pluripotent stem cells -- 19 Dysfunction and restoration of endothelial cell communications in Pulmonary Arterial Hypertension: Therapeutic implications -- 20 Inflammatory Cytokines in the Pathogenesis of Pulmonary Arterial Hypertension -- 21 Genotypes and Phenotypes of Chinese Pediatric Patients with Idiopathic and Heritable Pulmonary Arterial Hypertension- Experiences from A Single Center -- 22 Fundamental Insight into Pulmonary Vascular Disease : Perspectives from Pediatric PAH in Japan -- 23 Risk stratification in paediatric pulmonary arterial hypertension -- 24 The Adaptive Right Ventricle in Eisenmenger Syndrome: Potential Therapeutic Targets for Pulmonary Hypertension -- 25 Impaired right coronary vasodilator function in pulmonary hypertensive rat assessed by in vivo synchrotron microangiography -- 26 Relationship between mutations in ENG and ALK1 gene and the affected organs in hereditary hemorrhagic telangiectasia -- 27 A genetic analysis for patients with pulmonary arterial hypertension -- 28 Evaluation and visualization of right ventricle using three dimensional echocardiography -- 29 Pulmonary hypertension associated with post-operative Tetralogy of Fallot -- 30 Microscopic Lung Airway Abnormality and Pulmonary Vascular Disease Associated with Congenital Systemic to Pulmonary Shunt -- 31 Respiratory syncytial virus infection in infants with heart and lung diseases -- PART III: Ductus arteriosus: bridge over troubled vessels -- 32 Perspective for Part III -- 33 The ductus arteriosus, a vascular outsider, in relation to the pulmonary circulation -- 34 Molecular, genetic, and pharmacological modulation of the ductus arteriosus: KATP channels as novel drug targets -- 35 New mediators in the biology of the ductus arteriosus: Lessons from the chicken embryo -- 36 Constriction of the Ductus Arteriosus with KATP Channel Inhibitors -- 37 New insights on how to treat patent ductus arteriosus -- 38 Antenatal Administration of Betamethasone Contributes to Intimal thickening of the Ductus Arteriosus -- 39 Prostaglandin E-EP4-mediated fibulin-1 up-regulation plays a role in intimal thickening of the ductus arteriosus -- 40 Transcriptional profiles in the chicken ductus arteriosus during hatching -- 41 Inhibition of Cyclooxygenase Contracts Chicken Ductus Arteriosus -- 42 Prostaglandin E2 receptor EP4 inhibition constricts the rat ductus arteriosus -- 43 Dilatation of the Ductus Arteriosus by Diazoxide in Fetal and Neonatal Rats -- 44 The Effect of Long-term Administration of Plostaglandin E1 on Morphological Changes in Ductus Arteriosus -- 45 Significance of SGK1 as a protein kinase transcriptionally regulated by ALK1 signaling in vascular endothelial cells -- 46 Fabrication of Implantable Human Arterial Graft by Periodic Hydrostatic Pressure -- 47 Optimum preparation of Candida albicans cell wall extra (CAWE) for the mouse model of Kawasaki disease -- PART IV: Development and Regeneration of the Cardiovascular System -- 48 Perspective for Part IV -- 49 Advances in the second heart field -- 50 Novel cardiac progenitors for all components of the heart except for the right ventricle -- 51 Regional and TBX5-dependent gene expression in the atria: Implications for pulmonary vein development and atrial fibrillation -- 52 The Endocardium as a Master Regulator of Ventricular Trabeculation -- 53 The Role of Alternative mRNA Splicing in Heart Development -- 54 Progress in the Generation of Multiple Lineage Human-iPSC-derived 3D Engineered Cardiac Tissues for Cardiac Repair -- 55 Quantification of contractility in stem cell derived cardiomyocytes -- 56 A neurotrophic factor receptor GFRA2, a specific surface antigen for cardiac progenitor cells, regulates the process of myocardial compaction -- 57 Cardiac cell specification and differentiation by the defined factors -- 58 A Temporo-Spatial Regulation of Sema3c is Essential for Interaction of Progenitor Cells during Cardiac Outflow Tract Development -- 59 Spatiotemporally restricted developmental alterations in the anterior and secondary heart fields cause distinct conotruncal heart defects -- 60 Significance of transcription factors in the mechanisms of great artery malformations -- 61 The different c-kit expression in human induced pluripotent stem (iPS) cells between with feeder cells and without feeder cells -- 62 Establishment of induced pluripotent stem cells from immortalized B cell lines and their differentiation into cardiomyocytes -- 63 Establishment of an in vitro LQT3 model, using induced pluripotent stem cells from LQT3 patient-derived cardiomyocytes -- 64 Genetic Assessments for clinical courses of Left ventricle noncompaction -- 65 Elucidating the pathogenesis of congenital heart disease in the era of next-generation sequencing…”

Tabla de Contenidos: “…13.6 Routing Protocol -- 13.7 High Altitude Platforms (HAPs) -- 13.8 Connectivity Graph Metrics -- 13.9 Aerial Vehicle Network Simulator (AVENs) -- 13.10 Conclusion -- References -- 14 Artificial Intelligence in Logistics and Supply Chain -- 14.1 Introduction to Logistics and Supply Chain -- 14.2 Recent Research Avenues in Supply Chain -- 14.3 Importance and Impact of AI -- 14.4 Research Gap of AI-Based Supply Chain -- References -- 15 Hereditary Factor-Based Multi-Featured Algorithm for Early Diabetes Detection Using Machine Learning -- 15.1 Introduction -- 15.2 Literature Review -- 15.3 Objectives of the Proposed System -- 15.4 Proposed System -- 15.5 HIVE and R as Evaluation Tools -- 15.6 Decision Trees -- 15.7 Results and Discussions -- 15.8 Conclusion -- References -- 16 Adaptive and Intelligent Opportunistic Routing Using Enhanced Feedback Mechanism -- 16.1 Introduction -- 16.2 Related Study -- 16.3 System Model -- 16.4 Experiments and Results -- 16.5 Conclusion -- References -- 17 Enabling Artificial Intelligence and Cyber Security in Smart Manufacturing -- 17.1 Introduction -- 17.2 New Development of Artificial Intelligence -- 17.3 Artificial Intelligence Facilitates the Development of Intelligent Manufacturing -- 17.4 Current Status and Problems of Green Manufacturing -- 17.5 Artificial Intelligence for Green Manufacturing -- 17.6 Detailed Description of Common Encryption Algorithms -- 17.6.1 Triple DES (3DES)-(Triple Data Encryption Standard) -- 17.7 Current and Future Works -- 17.8 Conclusion -- References -- 18 Deep Learning in 5G Networks -- 18.1 5G Networks -- 18.2 Artificial Intelligence and 5G Networks -- 18.3 Deep Learning in 5G Networks -- Conclusion -- References -- 19 EIDR Umpiring Security Models for Wireless Sensor Networks -- 19.1 Introduction -- 19.2 A Review of Various Routing Protocols -- 19.3 Scope of Chapter…”

Tabla de Contenidos: “…Concept 2.3 The formation and function of molecules depend on chemical bonding between atoms -- Covalent Bonds -- Ionic Bonds -- Weak Chemical Interactions -- Molecular Shape and Function -- Concept 2.4 Chemical reactions make and break chemical bonds -- Concept 2.5 Hydrogen bonding gives water properties that help make life possible on Earth -- Cohesion of Water Molecules -- Moderation of Temperature by Water -- Floating of Ice on Liquid Water -- Water: The Solvent of Life -- Acids and Bases -- 3 Carbon and the Molecular Diversity of Life -- Overview Carbon Compounds and Life -- Concept 3.1 Carbon atoms can form diverse molecules by bonding to four other atoms -- The Formation of Bonds with Carbon -- Molecular Diversity Arising from Variation in Carbon Skeletons -- The Chemical Groups Most Important to Life -- ATP: An Important Source of Energy for Cellular Processes -- Concept 3.2 Macromolecules are polymers, built from monomers -- The Synthesis and Breakdown of Polymers -- The Diversity of Polymers -- Concept 3.3 Carbohydrates serve as fuel and building material -- Sugars -- Polysaccharides -- Concept 3.4 Lipids are a diverse group of hydrophobic molecules -- Fats -- Phospholipids -- Steroids -- Concept 3.5 Proteins include a diversity of structures, resulting in a wide range of functions -- Amino Acid Monomers -- Polypeptides (Amino Acid Polymers) -- Protein Structure and Function -- Concept 3.6 Nucleic acids store, transmit, and help express hereditary information -- The Roles of Nucleic Acids -- The Components of Nucleic Acids -- Nucleotide Polymers -- The Structures of DNA and RNA Molecules -- Concept 3.7 Genomics and proteomics have transformed biological inquiry and applications -- DNA and Proteins as Tape Measures of Evolution -- 4 A Tour of the Cell -- Overview The Fundamental Units of Life…”

Tabla de Contenidos: “…3.17 Carbohydrate Metabolic Disorder and Diseases, Their Investigations -- 3.17.1 Diabetes Mellitus -- 3.17.2 Glycogen Storage Diseases -- 3.17.3 Galactosemia -- 3.17.4 Hereditary Fructose Intolerance -- 3.17.5 Mucopolysaccharidoses -- 3.17.6 Disorders of Pyruvate Metabolism -- 3.18 Summary of Carbohydrate Metabolism -- Exercises -- Multiple Choice Questions -- Chapter 4: Composition and Metabolism of Lipids -- 4.1 Introduction -- 4.1.1 Definitions -- 4.2 Classification of Lipids -- 4.2.1 Simple Lipids -- 4.2.2 Compound Lipids -- 4.2.3 Terpenoids and Steroids -- 4.2.4 Derived Lipids -- 4.3 Simple Lipids -- 4.3.1 Structure of Triacylglycerides -- 4.3.2 Fats -- 4.3.3 Waxes -- 4.4 Compound Lipids -- 4.4.1 Basic Structure of Phospholipids -- 4.4.2 Phospho lipid Structures -- 4.4.3 Non-phosphorylated Lipids -- 4.5 Terpenoids and Steroids -- 4.5.1 Terpenes -- 4.5.2 Steroids -- 4.5.3 Animal Sterols -- 4.5.4 Bile Acids -- 4.6 Derived Lipids -- 4.6.1 Fatty Acids -- 4.6.2 Eicosanoids -- 4.7 Qualitative Test of Lipids -- 4.8 Metabolism of Fatty Acids -- 4.8.1 Fatty Acids Transport into Mitochondria -- 4.8.2 Fatty Acids Oxidation -- 4.8.3 β-Oxidation of Saturated Fatty Acids -- 4.9 Biosynthesis of Fatty Acids -- 4.9.1 Difference Between Fatty Acid Synthesis and b-oxidation Pathways -- 4.9.2 Digestion and Transport of Fatty Acids -- 4.10 Cholesterol Metabolism -- 4.10.1 Biosynthesis of Cholesterol -- 4.10.2 Regulation of Cholesterol Biosynthesis -- 4.10.3 Transport of Cholesterol -- 4.10.4 Catabolism of Cholesterol -- 4.11 Classification of Plasma Lipoproteins and the Systemic Pathway of Lipids -- 4.12 Biosynthesis of Triacylglycerols -- 4.12.1 Triacylglycerol Metabolism in the Intestines, Liver and Mammary Glands -- 4.13 Metabolic Disorder of Lipids -- Exercises -- Multiple Choice Questions -- Chapter 5: Composition and Metabolism of Amino Acids and Proteins…”

“…Although the conflict culture of the dualistic system wasn't strong enough against confessional polarization, the cooperation of the Habsburg dynasty with the court-oriented nobility from the provinces of the hereditary lands proved to be such a successful model of governmental organization that in the Austrian-Bohemian 'Dyarchy' it was continued on a larger scale. …”

“…Topics such as the extracellular matrix in hereditary diseases, reproduction, cancer, muscle, and tissue engineering applications, and diverse roles for integrins, are included in this collection…”

“…It also covers Drosophila role in ubiquitin-carboxyl-terminal hydrolase-L1 (UCH-L1) protein, eye development, anti-dUCH antibody, neuropathy target esterase (NTE), organophosphorous compound-induced delayed neuropathy (OPIDN) and hereditary spastic paraplegia (HSP). It also includes substrate specificities, kinetic parameters of recombinant glutathione S-transferases E6 and E7 (DmGSTE6 and DmGSTE7), detoxification and insecticidal resistance and antiviral immunity in Drosophila…”

“…Moreover this is a series of descriptions about civil laws, valid for different hereditary provinces of the Habsburg Monarchy, which should then serve as basic sources in regard of the project of an unified Civil Code, which was provided as a common law for the entire monarchy instead of particular laws in different provinces. …”

“…Originally the status was granted for a limited time, however, by the seventeenth century it had become hereditary. Beginning in the 1750s foreign descendants of Chinese origin were singled out and reclassified as imperial subjects (hwangjoin), giving them the right to participate in the palace-sponsored Ming Loyalist rituals. …”

“…Point mutations in gap junction channels have been found to cause hereditary diseases like the congenital deafness or the Charcot-Marie-Tooth neuropathy but the exact molecular mechanisms of gap junction malfunction from most of the mentioned illnesses are not fully understood. …”